Chronic Helicobacter pylori-assotiated infection in children, paracellular permeability of the gastric mucosa and food allergy
DOI:
https://doi.org/10.22141/2224-0551.14.1.2019.157879Keywords:
Helicobacter pylori, children, food allergy, claudins, tight junctions, paracellular permeabilityAbstract
Currently, issues related to tight junction dysfunction, are one of the most relevant and studied in scientific world. In spite of significant achievements, many problems are still open today. This review discusses the molecular structure and regulation of stomach tight junctions, the relationship between Helicobacter pylori and food allergy in children and the possibility of pharmacological regulation of paracellular permeability of the gastric mucosa. To write the article, information was searched using Scopus, Web of Science, MEDLINE, PubMed and Google Scholar databases.References
MOH of Ukraine. Uniform clinical protocol of the medical care for children with gastroesophageal reflux disease: Order 59 from 29.01.2013. Available from: https://zakon.rada.gov.ua/rada/show/v0059282-13. (in Ukrainian).
Abaturov OE, Gerasimenko OM. Modulation of the activity of TLR4 epithelial cells of the gastric mucosa during Helicobacter pylori infection. Sovremennaya pediatriya. 2009;(28):141-146. (In Ukrainian).
Abaturov OE, Gerasimenko ON, Zavhorodnia NYu. Hronicheskie CagA(+)Helicobacter pylori-associirovannye gastrity u detej [Chronic CagA (+) Helicobacter pylori-associated gastritis in children]. Monograph. Kharkov: SAM; 2014. 412 p. (in Russian).
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Copyright (c) 2019 O.E. Abaturov, A.E. Lykova

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